fried loss-of-function mutants were first identified in a clonal screen for oogenesis defects. Subsequently, we found that fried mutants exhibit behavioral defects during larval stages and eventually die as late larvae or early pupae. Mapping, sequencing and rescue of fried mutants showed that fried encodes HEATR2, a protein that others have shown to be required for ciliary axoneme formation and to be expressed in mechanosensory neurons in embryos (Diggle, et al., 2014). Here we present the mRNA expression pattern of fried/HEATR2 in larval and adult tissues based on mRNA in situ hybridization using an mRNA null allele as a negative control. We show that fried/HEATR2 mRNA is strongly expressed in salivary glands and adult egg chambers along with an array of other tissue types. We also discuss loss of function phenotypes in fried larvae and pupae.